*Corresponding author:Jonathan L Ajah, Specialty Registrar in Surgery, Jos University Teaching Hospital, Nigeria
Received:October 04, 2018; Published: October 15, 2018
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After surgical operation or trauma, postoperative hyperglycaemia develops as a result of increased glucose production combined with decreased glucose uptake in peripheral tissues [1-2]. This is largely as a result of insulin resistance which is transiently induced within the stressed patient. It has been suggested that mechanisms for this phenomenon include the action of pro-inflammatory cytokines and the decreased responsiveness of insulin-regulated glucose transporter proteins [1-2].