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ISSN: 2574 -1241
ISSN: 2574 -1241
Received:October 04, 2018; Published: October 15, 2018
DOI: 10.26717/BJSTR.2018.10.001895
After surgical operation or trauma, postoperative hyperglycaemia develops as a result of increased glucose production combined with decreased glucose uptake in peripheral tissues [1-2]. This is largely as a result of insulin resistance which is transiently induced within the stressed patient. It has been suggested that mechanisms for this phenomenon include the action of pro-inflammatory cytokines and the decreased responsiveness of insulin-regulated glucose transporter proteins [1-2].
Introduction | Background & Review of Literature| References|
International Association of Landscape Archaeology, Czech Glass Society, Czech Republic
Department of Chemistry, Semenov Institute of Chemical Physics, USSR Academy of Sciences, Moscow, Russia
Pharmaco-kinetics, dynamics and Drug Metabolism, Touro College of Pharmacy, USA
Professor of Nuclear Medicine, Faculty of Medicine and Surgery, University of Milan, Milan, Italy
Clinical Radiologist (MD) - Department of RADIOLOGY, Cosenza Hospital, Cosenza, Italy
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