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ISSN: 2574 -1241
ISSN: 2574 -1241
Received:October 04, 2018; Published: October 15, 2018
DOI: 10.26717/BJSTR.2018.10.001895
After surgical operation or trauma, postoperative hyperglycaemia develops as a result of increased glucose production combined with decreased glucose uptake in peripheral tissues [1-2]. This is largely as a result of insulin resistance which is transiently induced within the stressed patient. It has been suggested that mechanisms for this phenomenon include the action of pro-inflammatory cytokines and the decreased responsiveness of insulin-regulated glucose transporter proteins [1-2].
Introduction | Background & Review of Literature| References|
International Association of Landscape Archaeology, Czech Glass Society, Czech Republic
Professor of Nuclear Medicine, Faculty of Medicine and Surgery, University of Milan, Milan, Italy
Associate Professor at Department of Breast and Thyorid Surgey, Chongqing General Hospital, China
Neurology, LA BioMed Research Institute, USA
Endocrinology & Metabolism, Diabetes and Nutrition, Edith Cowan University, Australia
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